Database : HANSEN Search on : DOENCAS DO SISTEMA NERVOSO PERIFERICO [Subject descriptor] References found : 83 [refine] Displaying: 1 .. 20   in format [Detailed]

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Id: 19646 Author: Negesse, Yohannes. Title: Staging nerve involvement in M. Leprae infection. Source: Int. J. Lepr;67(2):167-168, Jun., 1999. . Descriptors: Mycobacterium leprae/imunol Mycobacterium leprae/fisiol Doenças do Sistema Nervoso/fisiopatol Electronic Medium: http://hansen.bvs.ilsl.br/textoc/revistas/intjlepr/1999/pdf/v67n2/v67n2cor03.pdf / en Location: BR191.1

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Id: 18750 Author: Jordy, Ceme Ferreira; Belda, Walter. Title: Considerações sôbre a condução nervosa periférica na hanseníase. Source: Rev. bras. Leprol;37(1/4):61-69, 1970. ^bilus, ^btab. Abstract: Em 16 pacientes de hanseníase, com sinais clínicos da neuropatia periférica, os autores estudaram o tempo de condução do impulso nervoso em 38 territórios, 22 correspondentes ao nervo ulnar e 16 ao nervo poplíteo lateral. Os valores encontrados se situam entre os valores classicamente admitidos como normais, ao contrário do que acontece em outras formas de neuropatias crônicas. A origem e o mecanismo destes achados são discutidos, supondo os autores que sua explicação se deve à ativação das fibras periféricas por um processo compressivo-isquêmico, determinado pela instalação da lesão leprótica do nervo. Recomendam a medida do tempo de condução do impulso nervoso como auxiliar do diagnóstico diferencial da neurite leprótica com outras neuropatias periféricas. (AU)^ipt. Descriptors: Neurite/diag Neurite/fisiopatol Hanseníase/compl Hanseníase/diag Hanseníase/fisiopatol Doenças do Sistema Nervoso Periférico/compl Doenças do Sistema Nervoso Periférico/fisiopatol Limits: Humanos Masculino Feminino Electronic Medium: http://hansen.bvs.ilsl.br/textoc/revistas/brasleprol/1970/PDF/v37n1-4/v37n1-4a05.pdf / pt Location: BR191.1

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Id: 18722 Author: Jordy, Ceme F; Belda, Walter; Manzolli, Silvio. Title: Transtornos neurológicos associados ao mal perfurante plantar leprótico. Source: Rev. bras. Leprol;34(1/4):45-51, jan.-dez. 1966. ^btab. Abstract: Os autores analisam os distúrbios neurológicos sensitivos, motores e tróficos em 100 pacientes de lepra, 32 dos quais portadores de mal perfurante plantar. Concluem ser o m.p.p. complicação tardia do processo patológico hanseniano que se manifesta tôda vez que profundas modificações sensitivas, motoras e tróficas, devidas principalmente a transtornos da inervação profunda dos pés, alteram a arquitetura dos segmentos distais dos membros inferiores. (AU)^ipt. Descriptors: Úlcera do Pé/compl Úlcera do Pé/diag Úlcera do Pé/fisiopatol Doenças do Sistema Nervoso/compl Doenças do Sistema Nervoso/diag Doenças do Sistema Nervoso/fisiopatol Electronic Medium: http://hansen.bvs.ilsl.br/textoc/revistas/brasleprol/1966/PDF/v34n1-4/v34n1-4a05.pdf / pt Location: BR191.1

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Id: 18210 Author: Echevarría, José Ramón Gómez; Cervera, Fatima Moll. Title: Avaliação neurologica completa na neuropatia hanseniana / Complete neurologica assessment in Hansen´s disease Source: In: Anon.Congresso Internacional de Hanseniase, 16. Salvador, Artes Graficas Federico Domenech, Ago.2002. p.63-74tab. Conference: Present in: Congresso Internacional de Lepra, 16, Salvador, 4-9 agosto 2002. Abstract: Dada a importância da afectação neurológica da Doença de Hansen, os autores tentam descrever neste artigo a forma de realizar uma exploração neurológica completa. Para efeito, são focados os pontos fundamentais desta prática, como a palpação e identificação dos nervos periféricos engrossados, o estudo da sensibilidade superficial e profunda, a exploração da mobilidade voluntária, o exame dos reflexos e a avaliação dos processos neutróficos secundários ao trauma. Devemos destacar que, na maioria das situações, para o diagnóstico da doença e fundamentalmente no trabalho de campo, realizaremos uma exploração baseada em todos estes pontos, embora muito mais simplificada. Como anexo do artigo serão apresentados os resultados obtidos com a exploração neurológica de 71 doentes controlados no Sanatório Fontilles. (AU). Descriptors: DOENCAS DO SISTEMA NERVOSO PERIFERICO/compl DOENCAS DO SISTEMA NERVOSO PERIFERICO/diag DOENCAS DO SISTEMA NERVOSO PERIFERICO/fisiopatol DOENCAS DO SISTEMA NERVOSO PERIFERICO/reabil NERVO TIBIAL/les NERVO TIBIAL/fisiol NERVO TIBIAL/fisiopatol NERVO TIBIAL/cirurg NERVO FACIAL/anorm NERVO FACIAL/fisiopatol NERVO FACIAL/cirurg HANSENIASE/compl HANSENIASE/diag HANSENIASE/reabil HANSENIASE/cirurg Limits: ESTUDO COMPARATIVO HUMANO Location: BR191.1; WC335.3, C76

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Id: 18013 Author: Organização Mundial da Saúde*. Title: Neuropatias Periféricas Peripheral Nervous System Diseases- Source: São Paulo; s.n; 1980. 142 p. ilus. Descriptors: DOENCAS DO SISTEMA NERVOSO PERIFERICO/compl DOENCAS DO SISTEMA NERVOSO PERIFERICO/diag DOENCAS DO SISTEMA NERVOSO PERIFERICO/microbiol DOENCAS DO SISTEMA NERVOSO PERIFERICO/fisiopatol DOENCAS DO SISTEMA NERVOSO PERIFERICO/reabil DOENCAS DO SISTEMA NERVOSO PERIFERICO/terap - NEURÔNIOS/citol  NEURÔNIOS/patol  NEURÔNIOS/fisiol Limits: ESTUDO COMPARATIVO HUMANO Location: BR191.1; WL500, O14n

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Id: 18010 Author: Maffei, Walter Edgard Title: As bases da anátomo-patológicas da neuriatria e psiquiatria The anatomy-pathology bases of the neuriatis and psychiatry-v.1 Source: São Paulo; s.n; 1951. 490 p. ilus. Descriptors: DOENCAS DO SISTEMA NERVOSO PERIFERICO/compl DOENCAS DO SISTEMA NERVOSO PERIFERICO/diag DOENCAS DO SISTEMA NERVOSO PERIFERICO/fisiopatol DOENCAS DO SISTEMA NERVOSO PERIFERICO/reabil DOENCAS DO SISTEMA NERVOSO PERIFERICO/terap NEUROSSIFILIS/diag NEUROSSIFILIS/fisiopatol NEUROSSIFILIS/reabil MENINGES/anorm MENINGES/anat MENINGES/les MENINGES/fisiopatol Limits: ESTUDO COMPARATIVO HUMANO Location: BR191.1A; WL, M269b

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Id: 18008 Author: Asbury, Arthur K; Johnson, Peter C Title: Pathology of Peripheral Nerve ..- Source: San Francisco; s.n; 1978. 311 p. ilus, graf. Descriptors: NERVOS PERIFERICOS/anat NERVOS PERIFERICOS/citol NERVOS PERIFERICOS/les NERVOS PERIFERICOS/fisiopatol NERVOS PERIFERICOS/cirurg DOENCAS DO SISTEMA NERVOSO PERIFERICO/diag DOENCAS DO SISTEMA NERVOSO PERIFERICO/fisiopatol DOENCAS DO SISTEMA NERVOSO PERIFERICO/reabil Limits: ESTUDO COMPARATIVO HUMANO Location: BR191.1; WL500, A18p

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Id: 18007 Author: Aminoff, M J. Title: Infectious and Inflammatory Neuropathies / ? Source: In: Aminoff, M J.Electromyography in Clinical practici. Menlo Park, s.n, 1978. p.172-173. Descriptors: DOENCAS DO SISTEMA NERVOSO PERIFERICO/compl DOENCAS DO SISTEMA NERVOSO PERIFERICO/fisiopatol DOENCAS DO SISTEMA NERVOSO PERIFERICO/reabil HANSENIASE/compl HANSENIASE/fisiopatol HANSENIASE/cirurg Limits: ESTUDO COMPARATIVO HUMANO

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Id: 18005 Author: Aminoff, M J. Title: Infectious and Inflammatory Neuropathies / ? Source: In: Aminoff, M J.Electromyography in Clinical practici. Menlo Park, s.n, 1978. p.172-173. Descriptors: DOENCAS DO SISTEMA NERVOSO PERIFERICO/compl DOENCAS DO SISTEMA NERVOSO PERIFERICO/fisiopatol DOENCAS DO SISTEMA NERVOSO PERIFERICO/reabil HANSENIASE/compl HANSENIASE/fisiopatol HANSENIASE/cirurg Limits: ESTUDO COMPARATIVO HUMANO

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Id: 17966 Author: Izikson L; English JC; Zirwas MJ Title: The flushing patient: differential diagnosis, workup, and treatment The flushing patient: differential diagnosis, workup, and treatment-v.55 Source: s.l; s.n; 2006. 15 p. ilus. Abstract: Cutaneous flushing-a common presenting complaint to dermatologists, allergists, internists, and family practitioners-results from changes in cutaneous blood flow triggered by multiple conditions. Most cases are caused by very common, benign diseases, such as rosacea or climacterum, that are readily apparent after a thorough taking of history and physical examination. However, in some cases, accurate diagnosis requires further laboratory, radiologic, or histopathologic studies to differentiate several important clinicopathologic entities. In particular, the serious diagnoses of carcinoid syndrome, pheochromocytoma, mastocytosis, and anaphylaxis need to be excluded by laboratory studies. If this work-up is unrevealing, rare causes, such as medullary carcinoma of the thyroid, pancreatic cell tumor, renal carcinoma, and others, should be considered. LEARNING OBJECTIVE: At the completion of this learning activity, participants should be familiar with the mechanisms of flushing, its clinical differential diagnosis, the approach to establish a definitive diagnosis, and management of various conditions that produce flushing.(AU). Descriptors: DOENCAS DO SISTEMA NERVOSO/compl DOENCAS DO SISTEMA NERVOSO/diag DOENCAS DO SISTEMA NERVOSO/fisiopatol NEOPLASIAS CUTÂNEAS/compl NEOPLASIAS CUTÂNEAS/genet NEOPLASIAS CUTÂNEAS/fisiopatol - ACNE ROSACEA/compl  ACNE ROSACEA/diag  ACNE ROSACEA/fisiopatol  NEOPLASIAS/compl  NEOPLASIAS/fisiopatol Limits: ESTUDO COMPARATIVO HUMANO Location: BR191.1; 00323/cme

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Id: 17691 Author: Scollard, D. M; Adams, L. B; Gillis, T. P; Krahenbuhl, J. L; Truman, R. W; Williams, D. L Title: The continuing challenges of leprosy ..- Source: s.l; s.n; 2006. 44 p. ilus, tab. Abstract: Leprosy is best understood as two conjoined diseases. The first is a chronic mycobacterial infection that elicits an extraordinary range of cellular immune responses in humans. The second is a peripheral neuropathy that is initiated by the infection and the accompanying immunological events. The infection is curable but not preventable, and leprosy remains a major global health problem, especially in the developing world, publicity to the contrary notwithstanding. Mycobacterium leprae remains noncultivable, and for over a century leprosy has presented major challenges in the fields of microbiology, pathology, immunology, and genetics; it continues to do so today. This review focuses on recent advances in our understanding of M. leprae and the host response to it, especially concerning molecular identification of M. leprae, knowledge of its genome, transcriptome, and proteome, its mechanisms of microbial resistance, and recognition of strains by variable-number tandem repeat analysis. Advances in experimental models include studies in gene knockout mice and the development of molecular techniques to explore the armadillo model. In clinical studies, notable progress has been made concerning the immunology and immunopathology of leprosy, the genetics of human resistance, mechanisms of nerve injury, and chemotherapy. In nearly all of these areas, however, leprosy remains poorly understood compared to other major bacterial diseases. (AU). Descriptors: Antiinfecciosos/TU Proteínas de Bactérias/ME Vacinas Bacterianas Modelos Animais de Doenças Suscetibilidade à Doença/IM Resistência Bacteriana a Drogas Genes Bacterianos/GE Predisposição Genética para Doença Genoma Bacteriano Imunidade Celular Imunidade Natural/GE Hansenostáticos/PD/TU Hanseníase/*/DI/MI/TH Mycobacterium leprae/*/CH/DE/IP/PH Nervos Periféricos/MI Doenças do Sistema Nervoso Periférico/MI/PA Reação em Cadeia da Polimerase Research Support, N.I.H., Extramural Células de Schwann/IM/MI Limits: HUMANO ANIMAL CAMUNDONGOS SUPPORT, NON-U.S. GOV'T Location: BR191.1; 09365/S

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Id: 17634 Author: Brandsma, J. Wim. Title: Prevention and non surgical treatment of common impairments: an overview. Source: In: Schwarz, Richard; Brandsma, Wim.Surgical reconstruction rehabilitation in leprosy and other neuropathies. Kathmandu, Ekta Books, 2004. p.33-38ilus. Descriptors: DOENCAS DO SISTEMA NERVOSO PERIFERICO/diag DOENCAS DO SISTEMA NERVOSO PERIFERICO/fisiopatol DOENCAS DO SISTEMA NERVOSO PERIFERICO/cirurg ULCERA CUTÂNEA/diag ULCERA CUTÂNEA/fisiopatol ULCERA CUTÂNEA/cirurg NEUROPATIAS PERONEAIS/fisiopatol NEUROPATIAS ULNARES/fisiopatol Limits: ESTUDO COMPARATIVO HUMANO Location: BR191.1, S96s

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Id: 17632 Author: Brakel, Wim H. Van. Title: Peripheral nerve function assessment. Source: In: Schwarz, Richard; Brandsma, Wim.Surgical reconstruction rehabilitation in leprosy and other neuropathies. Kathmandu, Ekta Books, 2004. p.11-24ilus, tab. Descriptors: DOENCAS DO SISTEMA NERVOSO PERIFERICO/diag DOENCAS DO SISTEMA NERVOSO PERIFERICO/reabil DOENCAS DO SISTEMA NERVOSO PERIFERICO/cirurg PELE/patol PELE/fisiopatol SENSIBILIDADE TERMICA/fisiol - ATAXIA/fisiopatol  DOENCA DOS NEURÔNIOS MOTORES/fisiopatol Limits: ESTUDO COMPARATIVO HUMANO Location: BR191.1, S96s

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Id: 17631 Author: Schwarz, Richard. Title: General principles of reconstructive surgery for leprosy and other peripheral neuropathies. Source: In: Schwarz, Richard; Brandsma, Wim.Surgical reconstruction rehabilitation in leprosy and other neuropathies. Kathmandu, Ekta Books, 2004. p.1-9ilus, tab. Descriptors: HANSENIASE/reabil HANSENIASE/cirurg DOENCAS DO SISTEMA NERVOSO PERIFERICO/reabil DOENCAS DO SISTEMA NERVOSO PERIFERICO/cirurg DEFORMIDADES DA MAO/reabil DEFORMIDADES DA MAO/cirurg PELE/anat PELE/les PELE/cirurg - TRANSFERÊNCIA DE TENDAO/métodos  TRANSFERÊNCIA DE TENDAO/util Limits: ESTUDO COMPARATIVO HUMANO Location: BR191.1, S96s

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Id: 14105 Author: Saunderson, Paul; Gebre, Shibru; Desta, Ketsela; Byass, Peter; Lockwood, Diana N. J. Title: El modelo de neuropatía relacionado con la lepra en los paceintes AMFES de Etiopía: definiciones, factores de riesgo y resultado. Source: Fontilles - Revista de Leprología;22(6):601-631, Sep.-Dic. 2000. tab, graf. Abstract: El trabajo evaluación en el campo de MDT (AMFES) de ALERT empezó en 1988 con un control y seguimiento de los pacientes diez años después de finalizar el tratamiento (RFT). Este trabajo describe los hallazgos obtenidos en esta cohorte en relación a la neuropatía y deterioro neural. Se incluyen 594 nuevos casos de lepra, 300 multibacilares (MB) y 294 paucibacilares (PB). El 55% de los pecientes presentó algún grado de deterioro funcional neural (NFI) después de iniciar la multiterapia (MDT). La incidencia total para la neuropatía era de 39 episodios por 100 PYAR durante el primer año del diagnóstico, disminuyendo gradualmente hasta 12 episodios por 100 PYAR en el sexto año. En los pacientes sin deterioro en el momento del diagnóstico, la incidencia de neuropatía era de 25 episodios por 100 PYAA para casos MB y 11 por 100 PYAR para PB en el primer año; en el 33% de los casos MB cuyo primer episodio de neuropatía se presentó después del diagnóstico, el primer episodio después del primer año o después del intervalo pre-fijado de tratamiento. Se informó más detalladamente de 73 pacientes con neuropatía después del diagnóstico: 34 (47%) con sólo un nervio comprometido y entre ellos 25 (73%) con un único episodio agudo de neuropatía. Nueve (27%) presentaron más episodios. En 39 (53%) habían más de un nervio implicado y de entre ellos 16 (41%) con episodio agudo único, mientras el 23 (59%) presentaron más episodios. Se definen los términos neuropatía "crónica y recurrente" y se aplican en la descripción del modelo de neuropatía en los casos repetitivos. El tratamiento con esteroides consigue una recuperación total en el 88% de los nervios con neuropatía aguda en el grupo de pacientes sin deterioro al inicio del estudios, pero sólo el 51% en los que presenta neuropatía crónica o recurrente. El tiempo medio de recuperación de la neuropatía aguda era de aproximadamente seis meses, pero en algunos casos se presentó gradualmenteentre los dos a trea años. Resulta menos probable recuperación total de una neuropatía grave que de una leve o moderada. También se recuperaron el 42% de los nervios con neuropatía aguda no tratados con esteroides. el grupo de pacientes con deterioro muy crónico en el momento del diagnóstico, la plena recuperación de la función neural tuvo lugar en 87/374 (25%) de los nervios...(AU). Descriptors: HANSENIASE/clas HANSENIASE/etnol HANSENIASE/prev DOENCAS DO SISTEMA NERVOSO PERIFERICO/clas ETIOPIA/etnol Electronic Medium: http://www.ilsl.br Location: BR191.1

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Id: 13726 Author: Ulvi, H; Yoldas, T; Yigiter, R; Müngen, B Title: R-R interval variation and the sympathetic skin response in the assessment of the autonomic nervous system in leprosy patients ..- Source: s.l; s.n; 2003. 8 p. ilus, tab. Abstract: OBJECTIVES: The aim of this study was to evaluate possible autonomic nervous system (ANS) dysfunction in leprosy patients with the sympathetic skin response (SSR) and the heart rate (R-R) interval variation (RRIV) measurements which are easy and reliable methods for evaluation of autonomic functions. MATERIAL AND METHODS: We studied 37 lepromatous leprosy patients (mean age: 38 +/- 17 years, range 23-62 years, 20 females and 17 males) and 35 age-matched healthy subjects (mean age: 34.19 +/- 12.74 years, range 24-48 years, 20 females and 15 males). Non-invasive bedside tests (orthostatic test, Valsalva ratio), R-R interval variation (RRIV) during at rest and deep breathing, the SSR latency and amplitude from both palms, and nerve conduction parameters were studied in all the subjects. RESULTS: The mean values of RRIV in leprosy patients during at rest [mean RRIV in patients, 17.42 +/- 8.64 per cent vs controls, 22.71 +/- 3.77 per cent (P < 0.05)] and during deep breathing [mean RRIV in patients, 21.64 +/- 9.08 per cent vs controls, 30.70 +/- 5.99 per cent (P < 0.005)] was significantly lower compared with the controls. The mean latency of SSR in leprosy patients [mean SSR latency in patients, 1.72 +/- 1.13 ms vs controls, 1.30 +/- 0.41 ms (P < 0.05)] was significantly prolonged compared with the controls. The mean amplitude of SSR in leprosy patients [mean SSR amplitude in patients, 0.54 +/- 0.57 microV vs controls, 1.02 +/- 0.56 microV (P > 0.05)] was smaller compared with the controls, but this difference was not significant. The mean Valsalva ratio in leprosy patients [mean in patients, 1.11 +/- 0.13 vs controls, 1.16 +/- 0.07 (P > 0.05)] was smaller compared with the controls, but not statistically significant. The mean difference of systolic and diastolic blood pressure between supine rest and during standing in leprosy patients were higher compared with the controls [mean systolic pressure in patients, 7 +/- 6 mmHg vs controls, 6 +/- 8 mmHg (P > 0.05) and mean diastolic pressure in patients, 3 +/- 3 mmHg vs controls, 3 +/- 2 mmHg (P > 0.05)], but they did not reach statistical significance. Furthermore, lower RRIV and the prolonged SSR latencies in leprosy patients were closely correlated to some parameters of sensorimotor nerve conduction and each other [median nerve distal latency and RRIV, r = -0.67 (P < 0.05), ulnar nerve distal latency and RRIV, r = -0.59 (P < 0.05), RRIV and SSR latency,...(AU). Descriptors: DOENCAS DO SISTEMA NERVOSO AUTÔNOMO/diag DOENCAS DO SISTEMA NERVOSO AUTÔNOMO/fisiopatol PRESSAO ARTERIAL/fisiol ELETROCARDIOGRAFIA ELETROMIOGRAFIA RESPOSTA GALVÂNICA DA PELE/fisiol CORACAO/inerv FREQUÊNCIA CARDIACA/fisiol HANSENIASE TUBERCULOIDE/diag HANSENIASE TUBERCULOIDE/fisiopatol HANSENIASE VIRCHOWIANA/diag HANSENIASE VIRCHOWIANA/fisiopatol TEMPO DE REACAO/fisiol SISTEMA NERVOSO SIMPATICO/fisiopatol - EXAME NEUROLOGICO Limits: HUMANO MASCULINO FEMININO ADULTO MEIA-IDADE Electronic Medium: http://www.ilsl.br Location: BR191.1; 08994/s

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Id: 13717 Author: Crawford, C. L Title: The schizophrenic career of a "Monster Drug" ..- Source: s.l; s.n; 2003. 1 p. . Descriptors: DAPSONA/uso terap ERITEMA NODOSO/quimioter HANSENOSTATICOS/uso terap HANSENIASE/prev HANSENIASE VIRCHOWIANA/quimioter DOENCAS DO SISTEMA NERVOSO/ind quim PIODERMA GANGRENOSO/quimioter TALIDOMIDA/ef adv TALIDOMIDA/uso terap Limits: HUMANO Electronic Medium: http://www.ilsl.br Location: BR191.1; 09141/s

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Id: 13568 Author: Pearson, J. M;; Ross, W. F Title: Nerve involvement in leprosy--pathology, differential diagnosis and principles of management ..- Source: s.l; s.n; sep. 1975. 4 p. ilus. Abstract: Nerve damage in non-lepromatous leprosy and in "reversal" reactions is the result of an immune response to the presence of antigenic material derived from leprosy bacilli within nerves.(AU). Descriptors: DIAGNOSTICO DIFERENCIAL IMUNOSSUPRESSAO HANSENIASE/compl DOENCAS DO SISTEMA NERVOSO/diag DOENCAS DO SISTEMA NERVOSO/quimioter DOENCAS DO SISTEMA NERVOSO/etiol DOENCAS DO SISTEMA NERVOSO/patol CORTICOSTEROIDES/uso terap Limits: HUMANO Electronic Medium: http://www.ilsl.br Location: BR191.1; 00228/s

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Id: 13543 Author: Malaviya, G. N; Mukherjee, A; Ramu, G Title: Nerve abscess in lepromatous leprosy ..- Source: s.l; s.n; jan. 1982. 7 p. ilus, tab. Abstract: 6 cases of nerve abscesses developing in patients of lepromatous leprosy are reported. The cases reported in literature have been reviewed and the pathogenesis of abscesses formation is discussed.(AU). Descriptors: MYCOBACTERIUM LEPRAE NERVO MEDIANO/patol HANSENIASE/compl HANSENIASE/patol DOENCAS DO SISTEMA NERVOSO PERIFERICO/etiol DOENCAS DO SISTEMA NERVOSO PERIFERICO/patol NERVO ULNAR/patol ABSCESSO/etiol ABSCESSO/patol Limits: RELATO DE CASO HUMANO MASCULINO ADULTO ADOLESCENTE Electronic Medium: http://www.ilsl.br Location: BR191.1; 00912/s

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Id: 13541 Author: Lockwood, Diana N. J Title: Leprosy elimination - a virtual phenomenon or a reality? ..- Source: s.l; s.n; Jun. 2002. 3 p. ilus, mapas, graf. Abstract: Leprosy is a leading cause of neurological disability. The World Health Organization´s leprosy elimination campaign has treated 11 milion patients, but case numbers are still rising in the major countries where leprosy is endemic.New methods for diagnosis and treatment proposed by the WHO risk missing disease and undertreating patients, and an opportunity for implemeting evidence based policies may be missed. (AU). Descriptors: MEDICINA BASEADA EM EVIDÊNCIAS HANSENIASE/compl HANSENIASE/epidemiol HANSENIASE/prev DOENCAS DO SISTEMA NERVOSO/microbiol SAUDE MUNDIAL POLITICA DE SAUDE COOPERACAO INTERNACIONAL PREVALÊNCIA INCIDÊNCIA Limits: HUMANO Location: BR191.1; 05062/s

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